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Ained by an incomplete understanding of the pathophysiology of scleroderma and by the enrolment of patients in the terminal stages of the disease. It is of interest to note that the dermal fibrosis of the relaxin knockout mouse is rescued by exogenous relaxin at 6 months but not rescued by administration at 12 months of age (Samuel et al., 2005). One of the interesting aspects that have emerged is

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