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Expression of myocardiumspecific structural genes (connexin 43, troponin T, and HCN4 ion channel) (Formigli et al., 2009). Coronary infusion of C2C12 skeletal myoblasts overexpressing relaxin in conjunction with administration of exogenous relaxin starting at day 30 after infarction in rats improves myocardial viability in the infarcted area compared with myoblast therapy alone (Bonacchi et al., 2

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